OCULAR DRUG DELIVERY SYSTEM PPT/PDF

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Description

OCULAR DRUG
DELIVERY SYSTEM

Presented by

MOHANASANKAR V,
M.Pharmacy I Sem

Department of Pharmaceutics,
College of PharmaMcwywPyhw,a.r DmMualoGMuaiidxe.dc.Coomrmas Medical College, Chennai 1

 

SYLLABUS ………………………….(DDS)

✓Unit- 4

▪Ocular Drug Delivery Systems:

Barriers of Drug Permeation, Methods to overcome

barriers

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CONTENTS

▪ Introduction to ODDS
▪Anatomy and Physiology of eye
▪Disease and Disorders of Eye
▪Factors affecting Ocular absorption of drugs
▪ Intra Ocular Barriers
▪Methods to overcome barriers- Novel Ocular Formulations
▪Evaluation of ODDS
▪Conclusions
▪References

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INTRODUCTION TO ODDS

▪Ocular administration of drug is primarily associated with the
need to treat ophthalmic diseases.

• Eg.Glaucoma,Conjunctivitis, etc

▪Eye is the most easily accessible site for topical
administration of a medication.
▪ Ideal ophthalmic drug delivery must be able to sustain the

drug release and to remain in the vicinity of front of the eye
for prolong period of time.

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OCULAR DRUG DELIVERY SYSTEM
▪ The novel approach of drug delivery system in which drug can instilled on the

cull de sac cavity of eye is known has ocular drug delivery system.

▪ Ocular administration of drug is primarily associated with need to treat the
ophthalmic diseases.

▪ It is challenging due to the presence of anatomical and physiological barriers.

▪ These barriers can affect drug entry into the eye following multiple routes of
administration (e.g.. Topical, systemic, and injectable ).

▪ Topical administration in the form of eye drops is preferred for treating anterior
segment diseases, as it is convenient and provides local delivery of drugs.(poor
drug absorption and low bioavailability)

▪ To improve the bioavailability of drug novel drug delivery system are also
preferred.

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• The most commonly employed ophthalmic dosage forms are solutions,
suspensions, and ointments.

▪ Human eye

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▪COMPOSITION OF EYE:

Water – 98%, Solid -1.8%, Protein – 0.67%,

Other mineral element
sugar – 0.65%, NaCl – 0.66% sodium, potassium and

ammonia – 0.79%.

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ANATOMY OF EYE

HUMAN EYE
▪ Diameter 23mm
▪ Structure comprises of

three layers
1.Outer layer :
cornea( clear, transparent)
sclera(white, opaque)
2.Middle layer:
iris (anterior)
choroid (posterior)
ciliary body (intermediate)
3.Inner layer: Retina

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▪SCLERA:
▪ The protective outer layer(White of eye) of the eye
▪ White colored fibrous membrane surrounding the eyeball
▪ it maintain the shape of the eye.

▪CORNEA:
▪ The front of the sclera, is transparent, Circular, Bulgy epithelial membrane and

allow light to enter the eye. the cornea providing much of the eye’s focusing
power.

▪ Cornea composed of 5 layers Epithelium

Bowman’s Membrane

Stroma

Descemet’s Membrane

Endothelium

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The cornea has five main layers of cells:

1. Epithelium: The outer layer of the cells that acts as a barrier against
damage and infection

2. Bowman’s membrane: A thin, tough membrane

3. Stroma: Consist of collagen fibers and account for 90% of the
cornea’s thickness

4. Descemet’s membrane: A thin membrane of collagen and elastic
fiber

5. Endothelium: A layer very delicate cells that cannot regenerate and
are Responsible for maintaining partial corneal dehydration and
transparency

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▪ FLUID SYSTEM:
• AQUEOUS HUMOR

1. Secreted from blood through epithelium of the ciliary body.

2. Secreted in posterior chamber and transported to anterior
chamber.

• VITREOUS HUMOR

1. Secreted from blood through epithelium of the ciliary body.

2. Diffuse through the vitreous body.

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▪ LACRIMAL GLANDS:

Secrete tears and wash foreign bodies.

▪CHOROID: It is the second layer of the eye and lies between the
sclera and retina it contains the blood vessels that provide
nourishment to the outer layer of the retina.

▪RETINA: It is the inner most layer in the eye. It converts image into
electrical impulses that are sent along the optic nerve to the brain
where the images are interpreted.

▪MACULA: It is located in the back of the eye in the centre of the
retina. This area produces sharpest vision.

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DISEASES OF EYE

▪Common disease affecting the anterior segment of the eye
✓ Dry eye syndrome
✓ Glaucoma
✓Allergic conjunctivitis
✓Anterior uveitis
✓Cataract

• Prominent diseases disease affecting the posterior segment of the
eye
✓Age Related Macular Degeneration (AMD)
✓Diabetic retinopathy Macular Edema(DME)
✓Proliferative Vitreo Retinopathy (PVR)
✓Posterior Uveitis
✓Cytomegalovirus(CMV)

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FACTORS AFFECTING OCULAR ABSORPTION OF DRUGS

1. Lacrimal Fluid
2. Nasolacrimal drainage
3. Molecular size
4. Partition Coefficient
5. Protein Binding
6. Charge
▪ Lacrimal Fluid

• When the eye drops are instilled in the cul-de-sac, the drug solution gets
diluted with the lacrimal fluid.

• This coupled with continuous tear flow decreases the volume and
concentration of drug reaching the target sites.

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▪Nasolacrimal Drinage
• This drainage system is also responsible for reducing the contact

time of the drug solution with the corneal surface
▪Molecular size

• Small size particles like mannitol (mol.wt 182) can easily pass
through an intact cornea when compared to large sized particles
like insulin and dextran

▪Partition Coefficient
• Corneal membrane being lipophilic is highly permeable to lipophilic drugs

while hydrophilic drugs experience greater resistance from the epithelium for
penetration. It has been observed that drugs permeate through the epithelium
of the cornea via the following two major pathways,

(a) Movement of drug molecules through transcellular route which is a partition
controlled pathway.
(b) Passage of molecules through the intercellular spaces.

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▪Protein Binding
• Upon instillation of the drug solution, proteins in the lacrimal fluid

bind with the drug molecules.
• Only free or unbound drug molecules are able to undergo corneal

permeation.
• In the cornea, free drug molecules undergo drug protein interaction

which produces a lag time thus preventing the penetration of drugs
in the anterior chamber.

• Such protein-drug interactions have found to decrease the
pharmacological activity of the drug.

▪Charge
• Surface of the corneal epithelium is negatively charged and

hence it favours the absorption of positively charged drug
molecules.

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INTRA OCULAR BARRIERS

• After the drug has been topically instilled into the precorneal area of the
eye in the form of the eye drops, it get removed by the following
mechanisms which can act alone or in combination

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EPITHELIAL
CORNEAL

TIGHT JUNCTION
BARRIERS

BARRIERS

NON-CORNEAL CONJUCTIVAL
ANTERIOR BARRIERS BARRIERS
SEGMENT

DEUG DELIVERY
BARRIERS: REFLEX BLINKING

TEAR TURN OVER

PRE CORNEAL
BARRIERS

NASOLACRIMAL DRINAGE

POSTERIOR BLOOD
AQUEOUS

SEGMENT
BARRIER

DRUG DELIVERY METABOLISM IN OCULAR

BARRIERS TISSUE
BLOOD RETINAL

BARRIERS

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BARRIERS

 

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EPITHELIAL TIGHT JUNCTION BARRIERS

Corneal epithelium is the primary barrier

Stratified corneal epithelium consists of a basal layer of columnar
cells, two or three layers of wing cells and one or two outer layers

of squamous cells.

Superficial cells are surrounded by the intercellular tight junction
(zonula occludens).

There are four tight junction Proteins, zo-1, Cingulin, zo-2 and
Occluden

Corneal Epithelium – Resist absorption of Hydrophylic drugs
Stroma- Barrier to Lipophilic drugs

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Non-corneal route
▪Conjunctival Barriers

▪Non-corneal route by passes the cornea and involves movement
across conjunctiva and sclera.

• Drug upon reaching beyond the cornea is absorbed by small
capillaries and is transferred to the systemic circulation.

• This route is important especially for large and hydrophilic
molecules such as peptides, proteins and si RNA (small or short
interfering RNA).E.g. Gentamycin, Timolol maleate etc.,

• The conjunctiva is more permeable than cornea especially for
hydrophilic molecules due to much lower expression of tight
junction proteins relative to corneal epithelium.

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Pre Corneal Barriers
▪REFLEX BLINKING

• A normal eyedropper delivers 25-56µL of the topical
formulation.(average volume 39µL)

• However, an eye can transiently hold up to 30 µl, and the rest
is lost either by nasolacrimal drainage or reflex blinking (5-7
blinks/ min), significantly decrease the overall drug available
for therapeutic action.

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▪NASOLACRIMAL DRINAGE
• Normal tear volume is approx.

7-9 µl.
• Lower eye lid hold the maximum

volume of about 25-30 µl.
• When medication is instilled into

the cul-de-sac, the constant tear
volume increase, which initiated
rapid reflex blinking.

• To maintain constant tear volume,
most of the drugs flows into
lacrimal drainage system into
nasolacrimal duct with in 5 mins.

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▪ Such drainage continues until the normal resident tear volume of 7-10 µl is attained.

▪ Due to this almost 80% of the instilled dose is lost thereby decreasing the contact
time of the drug with the precorneal surface and hence its ocular bioavailability.

▪ Once the normal resident tear volume is attained, drug concentration in the
precorneal area further decreases rapidly due to absorption by cornea and
conjunctiva and also due to rapid tear turnover.

▪ It has been found that nasolacrimal drainage is the major route through which the
drug is eliminated almost immediately following ocular administration due to which
only negligible quantity of the drug reaches the desired tissues.

▪ Other demerit with nasolacrimal drainage is that the drug may get absorbed through
the nasal mucosa, reach the systemic circulation and may precipitate undesirable
adverse effects.

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▪However, studies have shown that the rate of nasolacrimal drainage is
directly proportional to the volume of the instilled drug solution.

▪When smaller volumes of ophthalmic drugs were instilled, nasolacrimal
drainage was found to be less thereby increasing the intraocular
bioavailability of the drug.

▪Hence it has been proposed that a reduction in the volume of drug
solution instilled by an eye dropper from 50-70 µl to almost 5-10 µl would
significantly increase the ocular bioavailability of the drug and hence its
pharmacological activity.

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▪TEAR TURNOVER
• A significant impediment to topical ocular drug delivery is tear

turnover.
• Following topical administration an increase in the volume of cul-de-

sac occurs that leads to reflex blinking and increased tear secretion
eventually resulting in rapid drug loss from the precorneal area.

• Loss the solution occur due to tear turnover and nasolacrimal
drainage until the tear volume in the conjunctiva cul-de-sac returns
to normal range (7-9 µL)

• The initial first order drainage rate of eye drops from the ocular
surface is 1.2 µl/min in humans and 0.5-0.7 µL/min in rabbit.

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▪METABOLISM IN OCULAR TISSUES:
• Drug containing aromatic hydrocarbons are metabolized in the

pigmented epithelium and ciliary body to their corresponding
epoxides and phenols, or further metabolized by other enzymes
present in the eye.

• Hayakawa demonstrated that poor absorption of peptide drugs
and insulin is due to the extensive metabolism during
conjunctival permeation in albino rabbits.

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ANTERIOR AND POSTERIOR SEGMENT Bar.

▪Blood aqueous barriers
• The Endothelium of the Iris / Ciliary blood vessels and non

pigmented ciliary epithelium.
• Both the cell layers express tight junction complexes and

prevent the entry of the solutes into the intra ocular
environment such as aqueous humor.

▪Blood retinal barriers
• Retinal Capillary endothelial cells (inner)
• Retinal pigment epithelial cells ( outer) restrict the entry of the

therapeutic agents from the blood into posterior segment

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METHODS TO OVERCOME INTRA OCULAR BARRIERS –
NOVEL OCULAR FORMULATIONS

▪Conventional ocular drug delivery systems are very
inefficient, so attempts are made to design Novel Ocular
Drug Delivery System which improve the

• Drug-cornea Contact time
• Increase the corneal permeability
• Increased site specificity

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METHODS TO OVERCOME BARRIERS

ALTERNATIVE DRUG DELIVERY ROUTES

NOVEL DRUG DELIVERY SYSTEM

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▪ALTERNATIVE DRUG DELIVERY ROUTES

✓INTRAVITREAL INJECTIONS

✓SUB CONJUNCTIVAL INJECTIONS

✓RETROBULBAR ROUTE

✓PERIBULBAR ROUTE

✓INTRACAMERAL ROUTE

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OCCULAR DOSAGE FORMS

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CONVENTIONAL OCULAR DOSAGE FORMS

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NOVEL OCULAR DOSAGE FORMS

▪ADVANTAGES
Novel ocular drug delivery systems have been developed with an
aim to provide the following advantages.
1. Accurate and constant rate of drug delivery.
2. Provide sustained and controlled delivery of drugs.
3. Increase the intraocular bioavailability by prolonging the

residence time of the drug in the precorneal space.
4. Specifically targets the desired ocular tissues, thus preventing

systemic drug delivery.
5. Overcomes the barriers to efficient delivery like lacrimation,

nasolacrimal drainage, conjunctival uptake etc.
6. Improve patient compliance and therapeutic efficacy of drugs.

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OPHTHALMIC INSERTS

▪Ocular inserts are defined as sterile, thin, multi
layered, drug-impregnated, solid or semisolid
consistency devices placed into the cul-de-sac or
conjunctival sac, whose size and shape are especially
designed for ophthalmic application.
▪This helps in enhancing drug residence time in the
precorneal space and thus increase ocular
bioavailability.

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▪ IDEAL CHARACTERISTICS
An ideal controlled release ophthalmic insert should possess
the following properties.
1. It should not cause any discomfort
2. It should be easy to handle and insert
3. It should not interfere with vision and exchange of oxygen
4. It should exhibit reproducible release kinetics
5. It should offer ease in sterilization
6. It should be stable and easy to manufacture.

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▪MERITS
• Accurate dosing
• Prolongs drug contact time → increase ocular bioavailability
• Release drug at constant rate
• No nasolacrimal drainage → no systemic absorption →reduction in

undesirable effects
• Reduced frequency of use
• Targeted to internal ocular tissue
• More shelf life than conventional DF.

▪DEMERITS
• Poor patient acceptance as it is difficult to apply
• Device interfere with Vision
• Expensive

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▪CLASSIFICATION
1.NON ERODIBLE INSERTS:

A) Ocusert
B) Hydrophilic Contact lens

2. ERODIBLE INSERTS:
A) Lacriserts
B) SODI
C) NODDS
D)Corneal Collagen Shields

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▪OCUSERTS
▪ The ocusert therapeutic system is a flat, flexible, elliptical device

designed to be placed in the inferior cul-de-sac between the
sclera and the eyelid and to release pilocarpine continuously at a
steady rate for 7 days.

▪ The device consists of three layers…

I. Outer layer: ethylene vinyl acetate copolymer layer.

II. Inner layer: pilocarpine gelled with alginate main polymer

III.A retaining ring: of EVA impregnated with titanium di oxide .

▪ The ocuserts available in two forms:

( pilo 20- 20µg/hour) (pilo 40- 40µg/hour)

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Merits Demerits

• Difficult In Handing And Insertion

• Expelled From The Eye
• Controlled Rate Of Drug Delivery

• Thet Must Removed After Dosing
Period

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▪HYDROPHILIC CONTACT LENSES
• These are circular shaped structures.
• Dyes may be added during polymerization.
• Drug incorporation depends on whether their structure is hydrophilic

or hydrophobic.
▪Drug release depends upon :

• Amount of drug
• Soaking time.
• Drug concentration in soaking solution.

▪ ADVANTAGES:
• No preservation.
• Size and shape

▪ DISADVANTAGES:
• Handling and cleaning
• Expensive

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▪ERODIBLE INSERTS
▪The solid inserts absorbs the aqueous tear fluid and gradually erode or

disintegrate.

▪The drug is slowly leached from the hydrophilic matrix.

▪These do not have to removed at the end of their use from the body
tissues.

▪They are classifies as three types:

1. Lacriserts

2. SODI ( soluble ocular drug inserts)

3. New Ophthalmic Drug Delivery System (NODDS)

4. Corneal Collagen Shields
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▪LACRISERT
▪Lacrisert is a non-medicated, sterile, rod-shaped erodible

insert which is made from hydroxypropyl cellulose.
▪ It is devoid of any preservative and is useful in the treatment

of dry eye syndrome.
▪ It is placed in the inferior fornix, where it gets hydrated to form

a hydrophilic film, which in turn hydrates the cornea.

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▪Soluble Ocular Drug Insert (SODI)
▪ It is a sterile, small, oval-shaped wafer weighing 15-16 mg

which is composed of polyacrylamide (impregnated with
drug).
▪ It is inserted in the inferior cul-de-sac, where it undergoes

hydration, softens in 10-15 sec. and acquires the shape of
globe of the eye.
▪The film converts into a viscous polymer mass after 10-15

min and eventually into a polymer solution within the next 30-
60 min.
▪ It enables once-a-day therapy for treating trachoma and

glaucoma.

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▪New Ophthalmic Drug Delivery System (NODDS)
▪ It is a sterile, preservative-free system that helps to
deliver a film loaded with water soluble drug to the eye. It is
about 5 cm in length and 6 mm in breadth.
▪Each NODDS is basically composed of three films, all of
which are made up of polyvinyl alcohol (PVA) but differ in
terms of concentration.

(i) Medicated Flag: It is a circular-shaped film whose 40%
weight is composed of drug.
(ii) Membrane Film: It is a thin, water-soluble film which helps
to attach the medicated flag to the handle film.
(iii) Handle Film: It is a thicker, water soluble film that is
provided with a paper backing for strength.

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▪CORNEAL COLLAGEN SHIELDS
▪A disposable, short-term therapeutic bandage lens for the cornea.
▪ It conforms to the shape of the eye, protects the corneal surface, and

provides lubrication as it dissolves.
▪ The shields are derived from bovine collagen and are 14.5 mm in

diameter with thickness 0.15-0.19 mm.
▪ Sterilized by gamma irradiation.
▪Disadvantages
▪ 1. It is not optically clear.
▪ 2. The collagen shield causes some discomfort.

▪Clinical uses
▪ 1. Wound healing.
▪ 2. Treatment of dry eye.

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PENETRATION ENHANCERS

▪Another approach to increase intra ocular bioavailability of
drug was to incorporate suitable penetration enhancers like
preservatives(benzalkonium chloride), bile salts, chelating
agents, surfactants etc.
▪Usage of benzalkonium chloride and Chlorhexidine gluconate

increased the penetration of fluorescein in a normal eye
▪Chronic use top. Medicaments containing penetration

enhancers cause endothelial degeneration.

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MUCOADHESIVES
▪ Bioadhesion is defined as a phenomenon in which two materials, at least one of

which is of biological origin, are held together for prolong period of time by means
of interfacial forces.

▪ An attachment between an artificial material and a biological surface, which could
be an epithelial tissue, mucous membrane or a tissue surface is known as
adhesive attachment.

▪ The external surface of the globe of eye is covered by a thin film of glycoprotein
called as mucin.

▪ This layer is secreted by the goblet cells and it lines the cornea and conjunctiva.
▪ Thus mucin is a part of the corneal tear film and is capable of absorbing almost

40-80 times its weight of water.
▪ These mucoadhesive agents bind with the corneal-conjunctival mucin via non-

covalent bonds.
▪ Cationic and anionic polymers exhibit better mucoadhesive activity than non-

ionic polymers.
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USE OF MUCOADHESIVES

INCREASE CONTACT TIME

INCREASE DRUG RELEASE
FROM DF

Eg. RETISERT IMPLANT – Used for CHRONIC NON-INFECTIOUS UVEITIS

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GELS

OPHTHALMIC DRUGS

ADVANTAGES DISADVANTAGES

Ease Administration Blurred Vision

Inexpensiveness Matted eyelid after use

Inc. Drug corneal surface Reduced patient Compliance
contact

Long duration of action

Reduced dosing frequency

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In-situ Gel Forming Systems

▪ Induce Phase transition of Gel in the Cul-de-sac.

Change in pH Change in Temperature Change in Ionic Strength

• Cellulose acetate • Polyoxamers are temp • Gellan gum is an anionic

Phthalate(free flowing sensitive polyols polysaccharide present in
solution undergo phase

liquid at undergo phase
transition to form Gel

pH 4.4) transition. state upon exposure to
• If it contact with tear • They are solution at monovalent or Divalent

fluid pH 7.4,undergo room temp. cation.

phase transition to • When instilled at body • When introduced in eye
Na+ ion in laciaml fluid

form Gel , thus temp (35ºC) undergo
induce gellation

Prolongs drug release phase transition to
form Gel

High conc of polymer needed and cause irritation
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VISCOUS SYSTEM

Reduced drug corneal contact in conventional ocular forms

Visocifying agents inc. residence time of drugs

Reduce the Nasolacrimal drinage

Eg . PVA, MC, HEC, HPMC, HPC

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NANOPARTICLES

Drug containing nanoparticles upon administration in suspension form accumulate
at the site of administration from which it gets released either via diffusion,

chemical reaction, etc.

This helps in increasing the drug-corneal surface contact and thus
ocular bioavailability.

The first commercial nanoparticle formulation for ocular delivery was Pipolex. It
was composed of pilocarpine-loaded nanoparticles made from

poly(methylmethacrylate acrylic acid) copolymer.

These nanoparticles were found to increase the drug activity.

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LIPOSOMES
Liposomes are microscopic sealed structures in which the central aqueous

compartment is enclosed by one or more membrane-like phospholipid bilayers

Liposomes are amphiphilic in nature, hence both hydrophilic and lipophilic drugs
can be incorporated in them.

Liposomes were first used to deliver idoxuridine suspension to rabbits to treat
herpes simplex keratitis which exhibited greater therapeutic efficacy than its

aqueous solution.

MERITS DEMERITS

Deliver drug at constant rate Have shorter shelf life

Protect from metabolizing enzyme Limited loading capacity
Difficulty in Sterilization of

Biodegradable and Non toxic
liposomal formulation

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HYDROPHILIC HEAD

HYDROPHOBIC TAIL

AQUEOUS INTERIOR

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INTRAVITREAL IMPLANTS

Systemic administration of drug for treatment of chronic intraocular
infection id associated with undesirable adverse effects.

Intravitreal implants or Depot devices release drug at zero order rates
and Prolong rate of drug release and decrease dosing frequency

Examples of commercially available SR ocular insert is VITRASET
used in treatment of Cytomegalovirus retinitis

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IONTOPHORESIS

Iontophoresis is a technique in which charged ions are driven
across the membrane under the influence of electric current.

It enables in local drug delivery, is non-invasive, facilitates the transfer of high
molecular weight charged ions and also reduces the risk of systemic adverse

effects

Several drugs have been investigated for ocular delivery of drugs in rabbits via
iontophoresis such as cefazolin, dexamethasone, fluorescein, gentamicin,

ketoconazole, ticarcillin etc

Its adverse effects like erythema, itching, choroidal and retinal burns, as well
as corneal epithelial oedema.

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EVALUATION OF ODDS
UNIFORMITY OF THICKNESS

UNIFORMITY OF WEIGHT

DRUG CONTENT UNIFORMITY

DETERMINATION OF SURFACE PH

PERCENT MOISTURE ABSORPTION

PERCENT MOISTURE LOSS

SWELLING INDEX

FOLDING ENDURANCE

IN-VITRO DRUG RELEASE STUDY

IN-VIVO DRUG RELEASE STUDY
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Uniformity of Thickness

This test is applicable to ocular films, inserts, lenses
etc.

Five films are selected from each batch and the
thickness of each film is measured using a

micrometer screw guage.

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Uniformity of Weight

Five films are selected from each batch and each film is
weighed on an electronic balance and the mean weight of

each formulation is noted.

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Drug content Uniformity

This test for ocular inserts require the use of simulated tear
fluid (STF) with the following composition and pH of 7.4.

Sodium chloride – 0.67g
Sodium bicarbonate – 0.2 g
Hydrated calcium chloride – 0.008 g
Purified water – q s. 100 g

Each dosage form is ground in a glass mortar and pestle
to which the tear fluid is added to obtain a suspension.

This is then filtered and the filtrate is subjected to
spectrophotometric analysis to determine the drug

content.
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Determination of Surface pH

In this, initially an agar solution is prepared by
dissolving suitable quantity of agar in simulated

tear fluid and then pouring it in a petri dish.

The dosage form is placed in the petri dish and left
for 5 hours to swell at room temperature.

After this time period, pH of the surface is
measured using an electrode.

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Percent Moisture Absorption

The dosage forms are initially weighed and placed in a
desiccator containing 100 ml of saturated solution of AICI.

Humidity is maintained at 79.5%.

Dosage forms are removed after 3 days and weighed again.

The percent (%) of moisture absorbed is determined by the
following formula.

% moisture absorbed = Final weight – Initial weight x 100
Initial weight

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Percent Moisture Loss

Dosage forms are initially weighed and then placed in a
desiccator containing anhydrous calcium chloride.

The dosage forms are removed after a period of 3 days,
reweighed and percent (%) moisture loss is calculated from

the following formula.

% moisture loss =Initial weight – Final weight x 100
Initial weight

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Swelling Index

For this test, three beakers are taken and each is filled with 4 ml
simulated tear fluid

Three dosage forms which have been previously weighed are
added to each beaker and left to swell for 5 min.

After this period, the dosage forms are removed and excess
water is removed by using a filter paper

These are weighed and the water is continued to be removed
until there is no increase in weight. Swelling index is then

calculated from this data.

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Folding Endurance

This test helps to determine the brittleness of the dosage form.

It measures the number of folds made at the same place which
is required to break the dosage form or to induce the appearance

of visible cracks.

First fold is made by folding the dosage form in the centre using
the fingers and thumbs, then opening it.

The process is continued until the appearance of cracks or
breakage of the dosage form.

Folding endurance value of a dosage form is the total number
of folding operations performed.

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In-vitro Drug Release Study

• (a) Bottle Method

• (b) Diffusion Method

• (c) Modified Rotating Basket Method

• (d) Modified Rotating Paddle Apparatus

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In-vivo Drug Release Study

• Rabbit is preferred as the experimental animal as it has a large eye
size and its anatomical and physiological ocular features are similar to
that of humans. Drug concentration is measured in several ocular
tissues of the rabbit such as lens, iris, cornea, retina, sclera,
aqueous and vitreous humour etc.
▪ IOP measured by Tonometer

▪ Tear fluid sampled in capillaries – placed near marginal tear strip of eyes –
capillaries filled by capillary forces – lacrimal fluid obtained is analysed for
drug content

▪ Drug content in aqueous humor is analysed by anesthetizing rabbit by
ketamine- 1ml syringe with 26 guage needle – withdraw 200 ml aq. humor

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REFERENCES

▪ Controlled and Novel Drug Delivery By N.K.Jain

▪ Novel Drug Delivery System by Y.W. Chein

▪ Drug Delivery and Targeting (Anya .M. Hillery)

▪Occular Drug Delivery System by Pankaj Verma (Slideshare)

▪Occular Drug Delivery : Present Innovations and Future Challenges; Vrinde

Gote, The Journal of Pharmacology and Experimental Technologies

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THANK YOU….

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